An increase in systemic vascular resistance (SVR) is typically noted in which type of shock?

In cardiogenic shock, an increase in systemic vascular resistance (SVR) is typically observed due to the heart's compromised ability to pump effectively, which leads to a decrease in cardiac output. This inadequacy prompts the body to activate compensatory mechanisms, including vasoconstriction, to maintain blood pressure and perfusion to vital organs. As a response, the peripheral blood vessels constrict, which raises the SVR.

Understanding the pathophysiology of cardiogenic shock highlights that it is primarily driven by heart function failure, resulting in compensatory increases in resistance to maintain systemic pressure, rather than inadequate volume or anaphylactic responses that would lead to different hemodynamic changes. For instance, hypovolemic shock is characterized by low blood volume, resulting in decreased SVR initially. In neurogenic and anaphylactic shock, there is typically vasodilation rather than the increased resistance seen in cardiogenic shock, as these conditions lead to systemic hypotension due to different underlying mechanisms.